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Family, friends, and teammates watched Derek Boogaard's descent to death, in slow motion. When his body was discovered in his Minneapolis apartment on May 13, 2011, his NHL career and his life had been punctuated by substance abuse, stints in rehabilitation centers, and violence on the ice. Once the NFL's most intimidating enforcer, it took a lot to take him down.

The coroner ruled Boogaard, affectionately known as the "Boogeyman" died from a mixture of oxycodone and alcohol. Those close to him knew he was no stranger to OxyContin and Percocet. Left alone to sleep after a night out on the town, by the time his brothers Aaron and Ryan checked on him, he was gone. Derek was probably dead as soon as he closed his eyes, said the coroner, according to The New York Times.

Death by accidental overdose was the headline, but months later researchers determined he suffered from a degenerative disease associated with repetitive brain trauma. Boogaard's brain showed signs of chronic traumatic encephalopathy, or C.T.E. A lot of it.

Life of the enforcer

Elite enforcers change the game simply by stepping onto the ice. They are the intimidators, the warriors, the bullies of the team. They clean up the messes and right the wrongs inflicted upon their teammates by opposing players. When the time is right, gloves come off, the game stops and all eyes are on the enforcers' bare-fisted fights. Broken noses, smashed faces and concussions, part of the job. Their success is not measured in goals, but in fights won or lost. And Boogaard was the best.

In ninth grade Boogaard stood 6 feet 4 inches tall, weighed 210 pounds and by age 16, used his size to gain entrance to the hockey world. A perpetually average student, he didn't feel he was good at much else. Drafted by the Minnesota Wild in 2001, he would stay with the franchise until 2010 when he was traded to the New York Rangers.

Skating and boxing lessons helped make him who he eventually became, the most feared man in professional hockey. At age 24, in his second season in the NHL, the mere sight of the 6-8 Boogaard was intimidating.

Fighting in the NHL is legal, usually resulting in an expected 5-minute penalty, although hits to the head during play are not. Boogaard and his fellow enforcers are typically on the ice for only a few minutes per game, which made his last four-year, $6.5 million contract with the New York Rangers a very lucrative one. But the price they pay is high.

The hidden costs

Along with the injuries come painkillers and in some cases, addiction. Boogaard spent time in rehabilitation centers trying to cope with the increasing amount of pills he took on any given day. His family tried to help and he was enrolled in the Substance Abuse and Behavioral Health Program financed by the NHL and its players union.

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Researchers say victims of C.T.E. can exhibit Alzheimer-like symptoms such as memory loss and behavioral changes, although they are two distinct diseases. Those close to Boogaard say he had changed in the two years before his death and his memory lapses were not uncommon. The total number of concussions he suffered is anyone's guess.

Boogaard's brain was flown to Boston where it was examined by Dr. Ann McKee, one of the co-directors of the Center for the Study of Chronic Encephalopathy. McKee found his disease more advanced than Bob Probert's, an NHL enforcer who spent 16 years in the league.

McKee has examined nearly 80 brains from former athletes including many football players, one as young as 19. Boogaard's brain adds another piece to the C.T.E. mystery, along with the others. What factors play a role in who will suffer from the disease and how fast it will progress once it begins is under investigation.

Medical consensus statement on C.T.E.

A concussion consensus statement2 recently released by the American College of Sports Medicine, the American Academy of Family Physicians, the American Academy of Orthopaedic Surgeons, the American Medical Society for Sports Medicine, the American Orthopaedic Society for Sports Medicine and the American Osteopathic Academy of Sports Medicine “Concussion (Mild Traumatic Brain Injury) and the Team Physician: A Consensus Statement – 2011 Update” includes a section on C.T.E. It states:

○ A progressive neurodegenerative disease (tauopathy) caused by total brain trauma, and is not limited to athletes who have reported concussions.

○ The incidence and prevalence is unknown.

○ Diagnosed only after death by distinctive immunoreactive stains of the brain for tau protein and is not the same disease as Alzheimer.

○ Typical signs and symptoms include a decline of recent memory and executive function, mood, and behavioral disturbances (especially depression, impulsivity, aggressiveness, anger, irritability, suicidal behavior, and eventual progression to dementia).

○ Initial signs and symptoms do not typically manifest until decades afthttps://ma received (ages 40–50 yr).

○ A small subset of individuals with chronic traumatic encephalopathy have developed chronic traumatic encephalomyopathy, a progressive motor neuron disease characterized by profound weakness, atrophy, spasticity, and fasciculation similar to amyotrophic lateral sclerosis.


Sources:

Punched Out: The Life and Death of a Hockey Enforcer - The New York Times series December 3-5, 2011

(2) Medicine & Science in Sports & Exercise: December 2011 - Volume 43 - Issue 12 - p 2412–2422 doi: 10.1249/MSS.0b013e3182342e64

Special Communications

Photos courtesy of Sports Legacy Institute

Questions/comments? Contact Jean Rickerson at This email address is being protected from spambots. You need JavaScript enabled to view it.

 

Autopsy results from four former Canadian Football League (CFL) players showed the presence of chronic traumatic encephalopathy (CTE) in two of the mens' brains. CTE is a degenerative brain disease studied extensively by Boston area researchers at the Center for the Study of Chronic Traumatic Encephalopahy. Recent findings have raised questions about a link between repetitive head trauma, such as that suffered by athletes in contact sports, and the development of Lou Gehrig's disease, or ALS, as well as CTE.

Preliminary results on the CFL players were released today by the Canadian Sports Concussion Project.

The brains of Bobby Kuntz, Jay Roberts, Peter Ribbins and Tony Proudfoot were examined by Drs. Charles Tator and Richard Wennberg as well as others involved in the research.

CBC sports.ca reports:

"Patients who suffer from CTE can experience memory impairment, emotional instability, erratic behavior, depression and problems with impulse control. The condition can also advance into dementia.

'There are still so many unanswered questions surrounding concussion and the long-term consequences of repeated head injuries,' Tator said. 'We are trying to determine why some athletes in contact sports develop CTE and others don't, as well as how many concussions lead to the onset of this degenerative brain disease Also, we need to develop tests to detect this condition at an early stage and to discover treatments.'

Kuntz died in February 2011 at age 79 after a long battle with Parkinson's disease and diffuse Lewy body disease — a condition that overlaps with Parkinson's and Alzheimer's disease. Roberts, 67, passed away in October 2010 after suffering from dementia and lung cancer.

The results from the autopsies done on Ribbins and Proudfoot did not show signs of CTE. Ribbins, a receiver and defensive back with the Winnipeg Blue Bombers, died at age 63 of Parkinson's disease. Proudfoot, an all-star defensive back with the Montreal Alouettes, lost his battle with amyotrophic lateral sclerosis (ALS) this year at 61."

Questions/comments? Contact Jean Rickerson at This email address is being protected from spambots. You need JavaScript enabled to view it.



Read more: https://www.cbc.ca/sports/football/story/2011/07/26/sp-cfl-players-study.html?ref=rss#ixzz1TF6e6HTU

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